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“Near death experience” is not neurologically “superior”

  Some neurologists pointed out in the early 1980s that “near death experience” has the typical characteristics of limbic lobe (referring to a part of the brain) syndrome, which can be explained by the release of endorphins and enkephalins in the brain. A neuroscientist proposed in 1983 that the release of endorphins can cause a joy or emotional “near death experience”, while allyloxymorphone can produce a “hell-like “near death experience”.” The first formal “near-death experience” neurological model was proposed in 1987, which included endorphins, neurotransmission factors of the limbic system and other brain parts.
  In 1989, neuroscientists proposed a neurophysiological model of “near death experience”, and believed that serotonin played an important role in producing “near death experience”. Studies have found that a kind of anesthetic-ketamine can induce a “near death experience.” Through intravenous injection of a certain dose of ketamine, all the common content of “near death experience” can be produced. In the 1990s, neuroscientists conducted research on the hallucinogen dimethyltryptamine and proposed a hypothesis: the pineal gland of the brain before death or in a dying state releases a large amount of dimethyltryptamine, which is ” The cause of the “near death experience” phenomenon.
  In 2006, a scientist passed cardiovascular intervention, causing 42 healthy volunteers to faint. After the incident, these volunteers reported seeing light, tunnels, meeting departed relatives, and visiting other worlds. In 2008, a neuroscientist proposed that the “near death experience” of clinically deceased patients is a psychiatric dysfunction syndrome caused by severe brain failure (caused by the cessation of cerebral blood circulation). Studies have also shown that hypercapnia (hypercapnia) in the blood can induce “near death experience” symptoms such as light, vision, and mysterious experiences. Some neuroscientists have also suggested that “near death experience” is a hallucination caused by brain hypoxia, drugs or brain damage.
  Zola, an anesthesiologist at George Washington University in the United States, and his team believe that the “near death experience” is caused by a surge in electrical activity caused by the depletion of oxygen in the brain before death. The surge is similar to that of a fully conscious person, but the former’s blood pressure is too low to detect, but it can still produce vivid images and feelings. The gradual loss of brain activity occurs within approximately 1 hour before death and is interrupted by a burst of brain activity that lasts from 30 seconds to 3 minutes. A study conducted by Zola’s team on 7 dying patients found that the increase in brain electrical activity of the patients occurred when the blood pressure was no longer detectable. Their “near death experience” may be the memory of the total synaptic memory, and the end of life. But potentially reversible hypoxemia is related. A study published in 2010 stated that the root of the “near death experience” is that the high concentration of carbon dioxide in the blood changes the chemical balance of the brain, allowing the brain to “see” things.
  The activity of the left temporal lobe of the brain will increase in patients with “near death experience”. Stimulation of the temporal lobe is known to induce hallucinations, “soul out of the body” and memory flashbacks. In an experiment on a patient, electrical stimulation of the left temporal lobe apical junction area caused the patient to have the illusion that another person approached her. In 2011, an article published in the well-known magazine “Scientific American” concluded, “Scientific evidence shows that all the characteristics of the’near death experience’ have the basis of normal brain function errors.”
  Some scholars claimed that some “near death” The “experience” case occurred when the EEG showed a flat line (that is, when the brain was no longer functioning). However, some scholars have noticed that EEG is not a reliable indicator of brain death, because it can only detect half of the activity of the cerebral cortex, and cannot observe the deeper cerebral cortex structure.
  Many people who have experienced “near death experience” regard it as evidence of the existence of “life after death” (“the afterlife”) or “consciousness after death”. Idealists claim that “near death experience” is evidence that “dematerialized consciousness” or “soul” and the body exist separately and independently. However, materialist scholars point out that although physiological factors such as brain damage, intracranial hypoxia, or hypercapnia are not sufficient to explain the full nature of the “near-death experience”, this does not overturn the scientific conclusion that only a living human body can produce consciousness. , Idealism is fundamentally untenable.
  Scientists who hold a negative view of idealism point out that so far there is not enough evidence to support the claim that life exists after death. However, some studies have found that even during periods of unconsciousness, the brain can still record sensory impressions. For example, in an experiment conducted in 1983, scientists used a vocabulary tape to test the memory of a group of anesthetized patients. After physical recovery, these patients were able to identify which words appeared in the vocabulary list that was played to them at the time, with a significantly higher proportion than the incident rate. Scientists explain this that even under full anesthesia, the brain still retains a part of the ability to store new information. The “auditory” content that accompanies the “visual” in the “near death experience” is likely to be related to this.
  Scientists who do not believe in “post-death consciousness” and “the afterlife” also point out that the inference that the brains of clinically dying patients completely ceases to function, and that the near-death experience is formed after the death of the brain is untenable. In fact, most of the brain activity is not performed during the patient’s cardiac resuscitation attempt, because it takes too much time to save lives. So there is a possibility that even if the EEG shows a flat line (that is, the patient is brain-dead), the patient still has brain activity, and these activities are through functional magnetic resonance imaging, positron emission tomography or computer-assisted Tomography can be detected, this is because-unless directly connected to the brain through surgery, otherwise the electroencephalograph mainly measures the surface activity of the cerebral cortex.
  In short, the mainstream scientific community still does not support the so-called “post-mortem consciousness”, let alone the existence of “soul”. In fact, although the scientific community is still controversial about concepts such as consciousness pointers, death standards, and “near death experience”, the mainstream view is that matter determines consciousness, and there is no consciousness without life.

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